Online Submission. Nevertheless, there was significant elastic recoil, leaving a residual peak-to-peak gradient of 10 mm Hg. Formerly, this type of occlusion was considered to have a congenital origin.
Hospital Universitario La Paz. However, based on findings obtained with new imaging techniques and histological studies, it has been postulated that the membranes are the sequelae of a thrombotic process.
Subsequently, balloons of increasing caliber were advanced through the Mullins sheath over the guidewire, dilating the obstruction in successive steps. The procedure was successful and there was no residual gradient Figure 3A and B. Abstract A 47-year-old man was diagnosed with primary antiphospholipid syndrome and Budd-Chiari syndrome membranous complete obstruction of the intrahepatic inferior vena cava , with edema and ascites refractory to medical treatment.
At the follow-up over two years later, the patient remains asymptomatic and has not required diuretic treatment. The angiographic study in this patient showed complete membranous occlusion of the inferior vena cava.
Percutaneous Recanalization by Angioplasty and Stenting. Num 11. Log in. Iberoamerican Cardiovascular Journals Editors' Network.
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Right-sided follow-up catheterization at 12 months after stent implantation showed no significant restenosis Figure 3C. Image Bank. The most frequent cause in Western countries is thrombotic occlusion in a hypercoagulable state. The occlusion was dilated with balloons of increasing size and was subsequently stented successfully. Treatment for Budd-Chiari syndrome varies according to the etiology and level of the obstruction. Budd-Chiari syndrome is a heterogeneous group of diseases characterized by occlusion of the hepatic veins or the inferior vena cava, producing portal hypertension.
The inferior vena cava membrane was punctured with a Brockenbrough needle under multidirectional fluoroscopic guidance via a transfemoral approach.